Postural stability differentiates “lower body” from idiopathic parkinsonism
Identifieur interne : 000405 ( Main/Corpus ); précédent : 000404; suivant : 000406Postural stability differentiates “lower body” from idiopathic parkinsonism
Auteurs : C. Trenkwalder ; W. Paulus ; S. Krafczyk ; M. Hawken ; W. H. Oertel ; Th. BrandtSource :
- Acta Neurologica Scandinavica [ 0001-6314 ] ; 1995-06.
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- KwdEn :
Abstract
Introduction ‐ Patients with an akinetic Parkinson syndrome of the lower extremities and a poor response to L‐DOPA have been described as “lower body Parkinsonism” (LBP). These patients are characterized by frequent fallings and poor balance. Methods ‐ We have studied body sway with static (force platform) and dynamic (Equitest) posturography in 11 LBP patients, 6 of them revealing deep white matter lesions on MRI and 10 patients with advanced Parkinson's disease (PD) and compared performance with 30 age‐matched controls. Results ‐ When standing on a fixed support the postural performance of both patient groups lay within the normal range. The balance of LBP patients worsened in the static testing in the conditions “eyes open on foam” (p <0.05) and “eyes closed on foam” (p < 0.0006, of 11 patients falling), whereas the balance of PD patients deteriorated only with “eyes closed on foam” (p <0.05). Dynamic posturography confirmed these results in 6 different sensory conditions, clearly distinguishing the more unstable LBP patients from PD patients during “standing, eyes closed, foot support sway referenced” (p < 0.005). Conclusion ‐ We conclude, that postural adjustments in LBP patients are more disturbed than those in PD patients and posturography can be an additional tool for the differential diagnosis of Parkinson syndromes with gait disorders.
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DOI: 10.1111/j.1600-0404.1995.tb00444.x
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ISTEX:F9F9E3D25202392D3D8F3A125C614DECDCD14F05Le document en format XML
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Krafczyk</name><affiliation><mods:affiliation>Department of Neurology Ludwig‐Maximilians‐University of Munich, Charing Cross Hospital, London</mods:affiliation></affiliation></author><author><name sortKey="Hawken, M" sort="Hawken, M" uniqKey="Hawken M" first="M." last="Hawken">M. Hawken</name><affiliation><mods:affiliation>Department of Neurology, Charing Cross Hospital, London</mods:affiliation></affiliation></author><author><name sortKey="Oertel, W H" sort="Oertel, W H" uniqKey="Oertel W" first="W. H." last="Oertel">W. H. Oertel</name><affiliation><mods:affiliation>Department of Neurology Ludwig‐Maximilians‐University of Munich, Charing Cross Hospital, London</mods:affiliation></affiliation></author><author><name sortKey="Brandt, Th" sort="Brandt, Th" uniqKey="Brandt T" first="Th." last="Brandt">Th. 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Trenkwalder</name><affiliation><mods:affiliation>Department of Neurology Ludwig‐Maximilians‐University of Munich, Charing Cross Hospital, London</mods:affiliation></affiliation></author><author><name sortKey="Paulus, W" sort="Paulus, W" uniqKey="Paulus W" first="W." last="Paulus">W. Paulus</name><affiliation><mods:affiliation>Department of Clinical Neurophysiology, University of Gottingen, Germany, Charing Cross Hospital, London</mods:affiliation></affiliation></author><author><name sortKey="Krafczyk, S" sort="Krafczyk, S" uniqKey="Krafczyk S" first="S." last="Krafczyk">S. Krafczyk</name><affiliation><mods:affiliation>Department of Neurology Ludwig‐Maximilians‐University of Munich, Charing Cross Hospital, London</mods:affiliation></affiliation></author><author><name sortKey="Hawken, M" sort="Hawken, M" uniqKey="Hawken M" first="M." last="Hawken">M. Hawken</name><affiliation><mods:affiliation>Department of Neurology, Charing Cross Hospital, London</mods:affiliation></affiliation></author><author><name sortKey="Oertel, W H" sort="Oertel, W H" uniqKey="Oertel W" first="W. H." last="Oertel">W. H. Oertel</name><affiliation><mods:affiliation>Department of Neurology Ludwig‐Maximilians‐University of Munich, Charing Cross Hospital, London</mods:affiliation></affiliation></author><author><name sortKey="Brandt, Th" sort="Brandt, Th" uniqKey="Brandt T" first="Th." last="Brandt">Th. Brandt</name><affiliation><mods:affiliation>Department of Neurology Ludwig‐Maximilians‐University of Munich, Charing Cross Hospital, London</mods:affiliation></affiliation></author></analytic><monogr></monogr><series><title level="j">Acta Neurologica Scandinavica</title><idno type="ISSN">0001-6314</idno><idno type="eISSN">1600-0404</idno><imprint><publisher>Blackwell Publishing Ltd</publisher><pubPlace>Oxford, UK</pubPlace><date type="published" when="1995-06">1995-06</date><biblScope unit="volume">91</biblScope><biblScope unit="issue">6</biblScope><biblScope unit="page" from="444">444</biblScope><biblScope unit="page" to="452">452</biblScope></imprint><idno type="ISSN">0001-6314</idno></series><idno type="istex">F9F9E3D25202392D3D8F3A125C614DECDCD14F05</idno><idno type="DOI">10.1111/j.1600-0404.1995.tb00444.x</idno><idno type="ArticleID">ANE444</idno></biblStruct></sourceDesc><seriesStmt><idno type="ISSN">0001-6314</idno></seriesStmt></fileDesc><profileDesc><textClass><keywords scheme="KwdEn" xml:lang="en"><term>Parkinson's disease</term><term>balance</term><term>gait disorder</term><term>lower body Parkinsonism</term><term>static/dynamic posturography</term></keywords></textClass><langUsage><language ident="en">en</language></langUsage></profileDesc></teiHeader><front><div type="abstract" xml:lang="en">Introduction ‐ Patients with an akinetic Parkinson syndrome of the lower extremities and a poor response to L‐DOPA have been described as “lower body Parkinsonism” (LBP). These patients are characterized by frequent fallings and poor balance. Methods ‐ We have studied body sway with static (force platform) and dynamic (Equitest) posturography in 11 LBP patients, 6 of them revealing deep white matter lesions on MRI and 10 patients with advanced Parkinson's disease (PD) and compared performance with 30 age‐matched controls. Results ‐ When standing on a fixed support the postural performance of both patient groups lay within the normal range. The balance of LBP patients worsened in the static testing in the conditions “eyes open on foam” (p <0.05) and “eyes closed on foam” (p < 0.0006, of 11 patients falling), whereas the balance of PD patients deteriorated only with “eyes closed on foam” (p <0.05). Dynamic posturography confirmed these results in 6 different sensory conditions, clearly distinguishing the more unstable LBP patients from PD patients during “standing, eyes closed, foot support sway referenced” (p < 0.005). Conclusion ‐ We conclude, that postural adjustments in LBP patients are more disturbed than those in PD patients and posturography can be an additional tool for the differential diagnosis of Parkinson syndromes with gait disorders.</div></front></TEI><istex><corpusName>wiley</corpusName><author><json:item><name>C. Trenkwalder</name><affiliations><json:string>Department of Neurology Ludwig‐Maximilians‐University of Munich, Charing Cross Hospital, London</json:string></affiliations></json:item><json:item><name>W. Paulus</name><affiliations><json:string>Department of Clinical Neurophysiology, University of Gottingen, Germany, Charing Cross Hospital, London</json:string></affiliations></json:item><json:item><name>S. Krafczyk</name><affiliations><json:string>Department of Neurology Ludwig‐Maximilians‐University of Munich, Charing Cross Hospital, London</json:string></affiliations></json:item><json:item><name>M. Hawken</name><affiliations><json:string>Department of Neurology, Charing Cross Hospital, London</json:string></affiliations></json:item><json:item><name>W. H. Oertel</name><affiliations><json:string>Department of Neurology Ludwig‐Maximilians‐University of Munich, Charing Cross Hospital, London</json:string></affiliations></json:item><json:item><name>Th. Brandt</name><affiliations><json:string>Department of Neurology Ludwig‐Maximilians‐University of Munich, Charing Cross Hospital, London</json:string></affiliations></json:item></author><subject><json:item><lang><json:string>eng</json:string></lang><value>static/dynamic posturography</value></json:item><json:item><lang><json:string>eng</json:string></lang><value>balance</value></json:item><json:item><lang><json:string>eng</json:string></lang><value>lower body Parkinsonism</value></json:item><json:item><lang><json:string>eng</json:string></lang><value>Parkinson's disease</value></json:item><json:item><lang><json:string>eng</json:string></lang><value>gait disorder</value></json:item></subject><articleId><json:string>ANE444</json:string></articleId><language><json:string>eng</json:string></language><abstract>Introduction ‐ Patients with an akinetic Parkinson syndrome of the lower extremities and a poor response to L‐DOPA have been described as “lower body Parkinsonism” (LBP). These patients are characterized by frequent fallings and poor balance. Methods ‐ We have studied body sway with static (force platform) and dynamic (Equitest) posturography in 11 LBP patients, 6 of them revealing deep white matter lesions on MRI and 10 patients with advanced Parkinson's disease (PD) and compared performance with 30 age‐matched controls. Results ‐ When standing on a fixed support the postural performance of both patient groups lay within the normal range. The balance of LBP patients worsened in the static testing in the conditions “eyes open on foam” (p >0.05) and “eyes closed on foam” (p > 0.0006, of 11 patients falling), whereas the balance of PD patients deteriorated only with “eyes closed on foam” (p >0.05). Dynamic posturography confirmed these results in 6 different sensory conditions, clearly distinguishing the more unstable LBP patients from PD patients during “standing, eyes closed, foot support sway referenced” (p > 0.005). Conclusion ‐ We conclude, that postural adjustments in LBP patients are more disturbed than those in PD patients and posturography can be an additional tool for the differential diagnosis of Parkinson syndromes with gait disorders.</abstract><qualityIndicators><score>7.696</score><pdfVersion>1.4</pdfVersion><pdfPageSize>576 x 828 pts</pdfPageSize><refBibsNative>true</refBibsNative><keywordCount>5</keywordCount><abstractCharCount>1367</abstractCharCount><pdfWordCount>4652</pdfWordCount><pdfCharCount>30070</pdfCharCount><pdfPageCount>9</pdfPageCount><abstractWordCount>212</abstractWordCount></qualityIndicators><title>Postural stability differentiates “lower body” from idiopathic parkinsonism</title><genre><json:string>article</json:string></genre><host><volume>91</volume><publisherId><json:string>ANE</json:string></publisherId><pages><total>9</total><last>452</last><first>444</first></pages><issn><json:string>0001-6314</json:string></issn><issue>6</issue><genre><json:string>Journal</json:string></genre><language><json:string>unknown</json:string></language><eissn><json:string>1600-0404</json:string></eissn><title>Acta Neurologica Scandinavica</title><doi><json:string>10.1111/(ISSN)1600-0404</json:string></doi></host><publicationDate>1995</publicationDate><copyrightDate>1995</copyrightDate><doi><json:string>10.1111/j.1600-0404.1995.tb00444.x</json:string></doi><id>F9F9E3D25202392D3D8F3A125C614DECDCD14F05</id><fulltext><json:item><original>true</original><mimetype>application/pdf</mimetype><extension>pdf</extension><uri>https://api.istex.fr/document/F9F9E3D25202392D3D8F3A125C614DECDCD14F05/fulltext/pdf</uri></json:item><json:item><original>false</original><mimetype>application/zip</mimetype><extension>zip</extension><uri>https://api.istex.fr/document/F9F9E3D25202392D3D8F3A125C614DECDCD14F05/fulltext/zip</uri></json:item><istex:fulltextTEI uri="https://api.istex.fr/document/F9F9E3D25202392D3D8F3A125C614DECDCD14F05/fulltext/tei"><teiHeader><fileDesc><titleStmt><title level="a" type="main" xml:lang="en">Postural stability differentiates “lower body” from idiopathic parkinsonism</title></titleStmt><publicationStmt><authority>ISTEX</authority><publisher>Blackwell Publishing Ltd</publisher><pubPlace>Oxford, UK</pubPlace><availability><p>WILEY</p></availability><date>1995</date></publicationStmt><sourceDesc><biblStruct type="inbook"><analytic><title level="a" type="main" xml:lang="en">Postural stability differentiates “lower body” from idiopathic parkinsonism</title><author><persName><forename type="first">C.</forename><surname>Trenkwalder</surname></persName><note type="correspondence"><p>Correspondence: C. Trenkwalder, Neurologische Universitaetsklinik Muenchen, Klinikum Grosshadern, Marchioninistr. 15, 8000 Muenchen 70, Germany</p></note><affiliation>Department of Neurology Ludwig‐Maximilians‐University of Munich, Charing Cross Hospital, London</affiliation></author><author><persName><forename type="first">W.</forename><surname>Paulus</surname></persName><affiliation>Department of Clinical Neurophysiology, University of Gottingen, Germany, Charing Cross Hospital, London</affiliation></author><author><persName><forename type="first">S.</forename><surname>Krafczyk</surname></persName><affiliation>Department of Neurology Ludwig‐Maximilians‐University of Munich, Charing Cross Hospital, London</affiliation></author><author><persName><forename type="first">M.</forename><surname>Hawken</surname></persName><affiliation>Department of Neurology, Charing Cross Hospital, London</affiliation></author><author><persName><forename type="first">W. H.</forename><surname>Oertel</surname></persName><affiliation>Department of Neurology Ludwig‐Maximilians‐University of Munich, Charing Cross Hospital, London</affiliation></author><author><persName><forename type="first">Th.</forename><surname>Brandt</surname></persName><affiliation>Department of Neurology Ludwig‐Maximilians‐University of Munich, Charing Cross Hospital, London</affiliation></author></analytic><monogr><title level="j">Acta Neurologica Scandinavica</title><idno type="pISSN">0001-6314</idno><idno type="eISSN">1600-0404</idno><idno type="DOI">10.1111/(ISSN)1600-0404</idno><imprint><publisher>Blackwell Publishing Ltd</publisher><pubPlace>Oxford, UK</pubPlace><date type="published" when="1995-06"></date><biblScope unit="volume">91</biblScope><biblScope unit="issue">6</biblScope><biblScope unit="page" from="444">444</biblScope><biblScope unit="page" to="452">452</biblScope></imprint></monogr><idno type="istex">F9F9E3D25202392D3D8F3A125C614DECDCD14F05</idno><idno type="DOI">10.1111/j.1600-0404.1995.tb00444.x</idno><idno type="ArticleID">ANE444</idno></biblStruct></sourceDesc></fileDesc><profileDesc><creation><date>1995</date></creation><langUsage><language ident="en">en</language></langUsage><abstract xml:lang="en"><p>Introduction ‐ Patients with an akinetic Parkinson syndrome of the lower extremities and a poor response to L‐DOPA have been described as “lower body Parkinsonism” (LBP). These patients are characterized by frequent fallings and poor balance. Methods ‐ We have studied body sway with static (force platform) and dynamic (Equitest) posturography in 11 LBP patients, 6 of them revealing deep white matter lesions on MRI and 10 patients with advanced Parkinson's disease (PD) and compared performance with 30 age‐matched controls. Results ‐ When standing on a fixed support the postural performance of both patient groups lay within the normal range. The balance of LBP patients worsened in the static testing in the conditions “eyes open on foam” (p <0.05) and “eyes closed on foam” (p < 0.0006, of 11 patients falling), whereas the balance of PD patients deteriorated only with “eyes closed on foam” (p <0.05). Dynamic posturography confirmed these results in 6 different sensory conditions, clearly distinguishing the more unstable LBP patients from PD patients during “standing, eyes closed, foot support sway referenced” (p < 0.005). Conclusion ‐ We conclude, that postural adjustments in LBP patients are more disturbed than those in PD patients and posturography can be an additional tool for the differential diagnosis of Parkinson syndromes with gait disorders.</p></abstract><textClass xml:lang="en"><keywords scheme="keyword"><list><head>Keywords</head><item><term>static/dynamic posturography</term></item><item><term>balance</term></item><item><term>lower body Parkinsonism</term></item><item><term>Parkinson's disease</term></item><item><term>gait disorder</term></item></list></keywords></textClass></profileDesc><revisionDesc><change when="1995-06">Published</change></revisionDesc></teiHeader></istex:fulltextTEI><json:item><original>false</original><mimetype>text/plain</mimetype><extension>txt</extension><uri>https://api.istex.fr/document/F9F9E3D25202392D3D8F3A125C614DECDCD14F05/fulltext/txt</uri></json:item></fulltext><metadata><istex:metadataXml wicri:clean="Wiley, elements deleted: body"><istex:xmlDeclaration>version="1.0" encoding="UTF-8" standalone="yes"</istex:xmlDeclaration><istex:document><component version="2.0" type="serialArticle" xml:lang="en"><header><publicationMeta level="product"><publisherInfo><publisherName>Blackwell Publishing Ltd</publisherName><publisherLoc>Oxford, UK</publisherLoc></publisherInfo><doi origin="wiley" registered="yes">10.1111/(ISSN)1600-0404</doi><issn type="print">0001-6314</issn><issn type="electronic">1600-0404</issn><idGroup><id type="product" value="ANE"></id><id type="publisherDivision" value="ST"></id></idGroup><titleGroup><title type="main" sort="ACTA NEUROLOGICA SCANDINAVICA">Acta Neurologica Scandinavica</title></titleGroup></publicationMeta><publicationMeta level="part" position="06006"><doi origin="wiley">10.1111/ane.1995.91.issue-6</doi><numberingGroup><numbering type="journalVolume" number="91">91</numbering><numbering type="journalIssue" number="6">6</numbering></numberingGroup><coverDate startDate="1995-06">June 1995</coverDate></publicationMeta><publicationMeta level="unit" type="article" position="0044400" status="forIssue"><doi origin="wiley">10.1111/j.1600-0404.1995.tb00444.x</doi><idGroup><id type="unit" value="ANE444"></id></idGroup><countGroup><count type="pageTotal" number="9"></count></countGroup><titleGroup><title type="tocHeading1">Original Articles</title></titleGroup><eventGroup><event type="firstOnline" date="2009-01-29"></event><event type="publishedOnlineFinalForm" date="2009-01-29"></event><event type="xmlConverted" agent="Converter:BPG_TO_WML3G version:2.3.5 mode:FullText source:HeaderRef result:HeaderRef" date="2010-04-06"></event><event type="xmlConverted" agent="Converter:WILEY_ML3G_TO_WILEY_ML3GV2 version:4.0.1" date="2014-03-15"></event><event type="xmlConverted" agent="Converter:WML3G_To_WML3G version:4.1.7 mode:FullText,remove_FC" date="2014-10-14"></event></eventGroup><numberingGroup><numbering type="pageFirst" number="444">444</numbering><numbering type="pageLast" number="452">452</numbering></numberingGroup><correspondenceTo>C. Trenkwalder, Neurologische Universitaetsklinik Muenchen, Klinikum Grosshadern, Marchioninistr. 15, 8000 Muenchen 70, Germany</correspondenceTo><linkGroup><link type="toTypesetVersion" href="file:ANE.ANE444.pdf"></link></linkGroup></publicationMeta><contentMeta><unparsedEditorialHistory>Accepted for publication September 14, 1994</unparsedEditorialHistory><countGroup><count type="referenceTotal" number="29"></count><count type="linksCrossRef" number="7"></count></countGroup><titleGroup><title type="main">Postural stability differentiates “lower body” from idiopathic parkinsonism</title></titleGroup><creators><creator creatorRole="author" xml:id="cr1" affiliationRef="#a1" corresponding="yes"><personName><givenNames>C.</givenNames><familyName>Trenkwalder</familyName></personName></creator><creator creatorRole="author" xml:id="cr2" affiliationRef="#a2"><personName><givenNames>W.</givenNames><familyName>Paulus</familyName></personName></creator><creator creatorRole="author" xml:id="cr3" affiliationRef="#a1"><personName><givenNames>S.</givenNames><familyName>Krafczyk</familyName></personName></creator><creator creatorRole="author" xml:id="cr4" affiliationRef="#a3"><personName><givenNames>M.</givenNames><familyName>Hawken</familyName></personName></creator><creator creatorRole="author" xml:id="cr5" affiliationRef="#a1"><personName><givenNames>W. H.</givenNames><familyName>Oertel</familyName></personName></creator><creator creatorRole="author" xml:id="cr6" affiliationRef="#a1"><personName><givenNames>Th.</givenNames><familyName>Brandt</familyName></personName></creator></creators><affiliationGroup><affiliation xml:id="a1"><unparsedAffiliation>Department of Neurology Ludwig‐Maximilians‐University of Munich, Charing Cross Hospital, London</unparsedAffiliation></affiliation><affiliation xml:id="a2" countryCode="DE"><unparsedAffiliation>Department of Clinical Neurophysiology, University of Gottingen, Germany, Charing Cross Hospital, London</unparsedAffiliation></affiliation><affiliation xml:id="a3"><unparsedAffiliation>Department of Neurology, Charing Cross Hospital, London</unparsedAffiliation></affiliation></affiliationGroup><keywordGroup xml:lang="en"><keyword xml:id="k1">static/dynamic posturography</keyword><keyword xml:id="k2">balance</keyword><keyword xml:id="k3">lower body Parkinsonism</keyword><keyword xml:id="k4">Parkinson's disease</keyword><keyword xml:id="k5">gait disorder</keyword></keywordGroup><abstractGroup><abstract type="main" xml:lang="en"><p>Introduction ‐ Patients with an akinetic Parkinson syndrome of the lower extremities and a poor response to L‐DOPA have been described as “lower body Parkinsonism” (LBP). These patients are characterized by frequent fallings and poor balance. <i>Methods</i> ‐ We have studied body sway with static (force platform) and dynamic (Equitest) posturography in 11 LBP patients, 6 of them revealing deep white matter lesions on MRI and 10 patients with advanced Parkinson's disease (PD) and compared performance with 30 age‐matched controls. <i>Results</i> ‐ When standing on a fixed support the postural performance of both patient groups lay within the normal range. The balance of LBP patients worsened in the static testing in the conditions “eyes open on foam” (<i>p</i> <0.05) and “eyes closed on foam” (<i>p</i> < 0.0006, of 11 patients falling), whereas the balance of PD patients deteriorated only with “eyes closed on foam” (<i>p</i> <0.05). Dynamic posturography confirmed these results in 6 different sensory conditions, clearly distinguishing the more unstable LBP patients from PD patients during “standing, eyes closed, foot support sway referenced” (<i>p</i> < 0.005). <i>Conclusion</i> ‐ We conclude, that postural adjustments in LBP patients are more disturbed than those in PD patients and posturography can be an additional tool for the differential diagnosis of Parkinson syndromes with gait disorders.</p></abstract></abstractGroup></contentMeta></header></component></istex:document></istex:metadataXml><mods version="3.6"><titleInfo lang="en"><title>Postural stability differentiates “lower body” from idiopathic parkinsonism</title></titleInfo><titleInfo type="alternative" contentType="CDATA" lang="en"><title>Postural stability differentiates “lower body” from idiopathic parkinsonism</title></titleInfo><name type="personal"><namePart type="given">C.</namePart><namePart type="family">Trenkwalder</namePart><affiliation>Department of Neurology Ludwig‐Maximilians‐University of Munich, Charing Cross Hospital, London</affiliation><description>Correspondence: C. Trenkwalder, Neurologische Universitaetsklinik Muenchen, Klinikum Grosshadern, Marchioninistr. 15, 8000 Muenchen 70, Germany</description><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">W.</namePart><namePart type="family">Paulus</namePart><affiliation>Department of Clinical Neurophysiology, University of Gottingen, Germany, Charing Cross Hospital, London</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">S.</namePart><namePart type="family">Krafczyk</namePart><affiliation>Department of Neurology Ludwig‐Maximilians‐University of Munich, Charing Cross Hospital, London</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">M.</namePart><namePart type="family">Hawken</namePart><affiliation>Department of Neurology, Charing Cross Hospital, London</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">W. H.</namePart><namePart type="family">Oertel</namePart><affiliation>Department of Neurology Ludwig‐Maximilians‐University of Munich, Charing Cross Hospital, London</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">Th.</namePart><namePart type="family">Brandt</namePart><affiliation>Department of Neurology Ludwig‐Maximilians‐University of Munich, Charing Cross Hospital, London</affiliation><role><roleTerm type="text">author</roleTerm></role></name><typeOfResource>text</typeOfResource><genre type="article" displayLabel="article"></genre><originInfo><publisher>Blackwell Publishing Ltd</publisher><place><placeTerm type="text">Oxford, UK</placeTerm></place><dateIssued encoding="w3cdtf">1995-06</dateIssued><edition>Accepted for publication September 14, 1994</edition><copyrightDate encoding="w3cdtf">1995</copyrightDate></originInfo><language><languageTerm type="code" authority="rfc3066">en</languageTerm><languageTerm type="code" authority="iso639-2b">eng</languageTerm></language><physicalDescription><internetMediaType>text/html</internetMediaType><extent unit="references">29</extent></physicalDescription><abstract lang="en">Introduction ‐ Patients with an akinetic Parkinson syndrome of the lower extremities and a poor response to L‐DOPA have been described as “lower body Parkinsonism” (LBP). These patients are characterized by frequent fallings and poor balance. Methods ‐ We have studied body sway with static (force platform) and dynamic (Equitest) posturography in 11 LBP patients, 6 of them revealing deep white matter lesions on MRI and 10 patients with advanced Parkinson's disease (PD) and compared performance with 30 age‐matched controls. Results ‐ When standing on a fixed support the postural performance of both patient groups lay within the normal range. The balance of LBP patients worsened in the static testing in the conditions “eyes open on foam” (p <0.05) and “eyes closed on foam” (p < 0.0006, of 11 patients falling), whereas the balance of PD patients deteriorated only with “eyes closed on foam” (p <0.05). Dynamic posturography confirmed these results in 6 different sensory conditions, clearly distinguishing the more unstable LBP patients from PD patients during “standing, eyes closed, foot support sway referenced” (p < 0.005). Conclusion ‐ We conclude, that postural adjustments in LBP patients are more disturbed than those in PD patients and posturography can be an additional tool for the differential diagnosis of Parkinson syndromes with gait disorders.</abstract><subject lang="en"><genre>Keywords</genre><topic>static/dynamic posturography</topic><topic>balance</topic><topic>lower body Parkinsonism</topic><topic>Parkinson's disease</topic><topic>gait disorder</topic></subject><relatedItem type="host"><titleInfo><title>Acta Neurologica Scandinavica</title></titleInfo><genre type="Journal">journal</genre><identifier type="ISSN">0001-6314</identifier><identifier type="eISSN">1600-0404</identifier><identifier type="DOI">10.1111/(ISSN)1600-0404</identifier><identifier type="PublisherID">ANE</identifier><part><date>1995</date><detail type="volume"><caption>vol.</caption><number>91</number></detail><detail type="issue"><caption>no.</caption><number>6</number></detail><extent unit="pages"><start>444</start><end>452</end><total>9</total></extent></part></relatedItem><identifier type="istex">F9F9E3D25202392D3D8F3A125C614DECDCD14F05</identifier><identifier type="DOI">10.1111/j.1600-0404.1995.tb00444.x</identifier><identifier type="ArticleID">ANE444</identifier><recordInfo><recordContentSource>WILEY</recordContentSource><recordOrigin>Blackwell Publishing Ltd</recordOrigin></recordInfo></mods></metadata><serie></serie></istex></record>Pour manipuler ce document sous Unix (Dilib)
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